Der Kampf gegen die internationale Pillenmafia > Kampf der internationalen Pillen-Mafia!

Christopher Lacharite Mueller, Email: clm314@hotmail.com, wants war!?

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Krant:
Marke: 73.000

Vrolliastar:
Mark 74000

Vrolliastar:
http://www.reversecanada.com/lookup/8193836899/

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Home » Lookup » (819) 383-6899

    Christopher Mueller
    January 8, 2018
    Unknown
    Canada

clm314 @ hotmail.com
Christopher Mueller
540 des Ursulines
Trois-Rivieres
Quebec
G9A 5B2
CANADA

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Nur noch "Christopher Mueller"?


https://ca.linkedin.com/people/search?firstName=++++Christopher&lastName=Mueller+Lacharite&trk=public_profile_people-search-bar_search-submit

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1 result for " Christopher Mueller Lacharite"

    Christopher Lacharité Mueller

    Bachelor of Science at UQTR

    Trois-Rivieres, QC
    UQTR
    Université Laval
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Vrolliastar:
If you ask me: this looks like he got a kick in his behind in 2012, the very year he dared to mess up with Allaxysians.

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Education

    Université Laval
    Master’s Degree Biophotonics  Non completed

    2010 – 2012
    MSc in biophotonics, mainly applied to neurobiology research. Non completed.MSc in biophotonics, mainly applied to neurobiology research. Non completed.
[*/quote*]


Further investigations show, that when he messed up, he still was at the university. So it is quite likely that making his crimes public cost him his academic career.

Revenge is on our side. Do not mess up with Allaxysians, or you are done.



https://ca.linkedin.com/in/christopher-lacharit%C3%A9-mueller-1b3b82130?trk=people-guest_profile-result-card_result-card_full-click

[*quote*]
Christopher Lacharité Mueller
Bachelor of Science at UQTR
Trois-Rivieres, Quebec, Canada3 connections
Join to Connect
UQTRUQTR

About

Perfectly bilingual with a neurobiology research background in an academic setting.

I'm looking for a full-time position in a pharmaceutical setting, either promoting or helping with the development of therapeutic solutions. That would allow me to help improve the quality of life of Canadians.
Experience

    UQTR
    Bachelor of Science
    UQTR

    Sep 2006 – Aug 2009  3 years

    Trois-Rivières, Québec, Canada
    BSc in Medical Biology, finished with 3.57 GPABSc in Medical Biology, finished with 3.57 GPA

Education

    Université Laval
    Master’s Degree Biophotonics  Non completed

    2010 – 2012
    MSc in biophotonics, mainly applied to neurobiology research. Non completed.MSc in biophotonics, mainly applied to neurobiology research. Non completed.

Licenses & Certifications

    Adwords fundamentals
    Google AdWords Certified
    Issued Oct 2016Expires Oct 2017
    See credentialExternal link

    Search Advertising
    Google AdWords Certified
    Issued Oct 2016Expires Oct 2017
    See credentialExternal link

Publications

    Partial dopamine depletion in MPTP-treated mice differentially altered motor skill learning and action control
    Behavioural Brain ResearchNovember 23, 2011
    Recent findings suggest that the neurotransmitter dopamine (DA) system plays a role in motor control and the acquisition of habits and skills. However, isolating DA-mediated motor learning from motor performance remains challenging as most studies include often severely DA-depleted mice. Using the neurotoxin 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP), we investigated the effect of various degrees of DA-depletion in mice on three tests of motor behaviors: the accelerating rotarod, wire suspension and pole tests. Three protocols were performed to decrease DA synthesis to various extents: 4 injections (i.p.) of 9 mg/kg in 1 day; 4 injections (i.p.) of 15 mg/kg in 1 day; or 5 injections (s.c.) of 30 mg/kg in 5 days. Severity of DA-depletion was assessed by the evaluation of tyrosine hydroxylase (TH) and dopamine transporter levels in the striatum using the Western blot technique. Mice were gathered into four different groups according their TH levels: mild, moderate, marked and severe. In these mice, the general motor abilities such as coordination, motion speed and muscular strength were relatively intact whereas impaired acquisition of skilled behavior occurred in mice with marked and severe reduction in TH levels. Marked and severely DA-depleted mice exhibited lower scores within the first trials of the first training day as well as a much slower progression in the following days on the accelerating rotarod. Based on these results, we conclude that the learning of a skilled behavior is more vulnerable to DA depletion than the DA-mediated control of motor activity.Recent findings suggest that the neurotransmitter dopamine (DA) system plays a role in motor control and the acquisition of habits and skills. However, isolating DA-mediated motor learning from motor performance remains challenging as most studies include often severely DA-depleted mice. Using the neurotoxin 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP), we investigated the effect of various degrees of DA-depletion in mice on three tests of motor behaviors: the accelerating rotarod, wire suspension and pole tests. Three protocols were performed to decrease DA synthesis to various extents: 4 injections (i.p.) of 9 mg/kg in 1 day; 4 injections (i.p.) of 15 mg/kg in 1 day; or 5 injections (s.c.) of 30 mg/kg in 5 days. Severity of DA-depletion was assessed by the evaluation of tyrosine hydroxylase (TH) and dopamine transporter levels in the striatum using the Western blot technique. Mice were gathered into four different groups according their TH levels: mild, moderate, marked and severe. In these mice, the general motor abilities such as coordination, motion speed and muscular strength were relatively intact whereas impaired acquisition of skilled behavior occurred in mice with marked and severe reduction in TH levels. Marked and severely DA-depleted mice exhibited lower scores within the first trials of the first training day as well as a much slower progression in the following days on the accelerating rotarod. Based on these results, we conclude that the learning of a skilled behavior is more vulnerable to DA depletion than the DA-mediated control of motor activity.

Other authors

    Chagniel L.Robitaille C.Bureau G.Cyr M.

See publicationExternal link
Age-dependent remodelling of inhibitory synapses onto hippocampal CA1 oriens-lacunosum moleculare interneurons.
Journal of PhysiologyAugust 8, 2011
Stratum oriens-lacunosum moleculare interneurons (O-LM INs) represent the major element of the hippocampal feedback inhibitory circuit, which provides inhibition to the distal dendritic sites of CA1 pyramidal neurons. Although the intrinsic conductance profile and the properties of glutamatergic transmission to O-LM INs have become a subject of intense investigation, far less is known about the properties of the inhibitory synapses formed onto these cells. Here, we used whole-cell patch-clamp recordings in acute mouse hippocampal slices to study the properties and plasticity of GABAergic inhibitory synapses onto O-LM INs. Surprisingly, we found that the kinetics of inhibitory postsynaptic currents (IPSCs) were slower in mature synapses (P26-40) due to the synaptic incorporation of the α5 subunit of the GABA(A) receptor (a5-GABA(A)R). Moreover, this age-dependent synaptic expression of a5-GABA(A)Rs was directly associated with the emergence of long-term potentiation at IN inhibitory synapses. Finally, the slower time course of IPSCs observed in O-LM INs of mature animals had a profound effect on IN excitability by significantly delaying its spike firing. Our data suggest that GABAergic synapses onto O-LM INs undergo significant modifications during postnatal maturation. The developmental switch in IPSC properties and plasticity is controlled by the synaptic incorporation of the a5-GABA(A)R subunit and may represent a potential mechanism for the age-dependent modifications in the inhibitory control of the hippocampal feedback inhibitory circuit.Stratum oriens-lacunosum moleculare interneurons (O-LM INs) represent the major element of the hippocampal feedback inhibitory circuit, which provides inhibition to the distal dendritic sites of CA1 pyramidal neurons. Although the intrinsic conductance profile and the properties of glutamatergic transmission to O-LM INs have become a subject of intense investigation, far less is known about the properties of the inhibitory synapses formed onto these cells. Here, we used whole-cell patch-clamp recordings in acute mouse hippocampal slices to study the properties and plasticity of GABAergic inhibitory synapses onto O-LM INs. Surprisingly, we found that the kinetics of inhibitory postsynaptic currents (IPSCs) were slower in mature synapses (P26-40) due to the synaptic incorporation of the α5 subunit of the GABA(A) receptor (a5-GABA(A)R). Moreover, this age-dependent synaptic expression of a5-GABA(A)Rs was directly associated with the emergence of long-term potentiation at IN inhibitory synapses. Finally, the slower time course of IPSCs observed in O-LM INs of mature animals had a profound effect on IN excitability by significantly delaying its spike firing. Our data suggest that GABAergic synapses onto O-LM INs undergo significant modifications during postnatal maturation. The developmental switch in IPSC properties and plasticity is controlled by the synaptic incorporation of the a5-GABA(A)R subunit and may represent a potential mechanism for the age-dependent modifications in the inhibitory control of the hippocampal feedback inhibitory circuit.

    Other authors
        Salesse C.Chamberland S.Topolnik L.See publicationExternal link

Languages

    English
    Native or bilingual proficiency

    French
    Native or bilingual proficiency

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Ayumi:
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